I was told in detox that my BAC was .6. What determines lethality to an individual and why?

Having never been informed of my BAC level, I was unaware of what it meant to have a level of .6. I remember this instance and recall being aware and conversational at the time. I am wondering what determines an individual's likelihood of having a lethal dose. I'm fairly young and am also wondering how significantly my liver might become damaged with regular episodes of heavy binges. As with smoking, is it possible for certain individuals to be resistant to ill effects/damage of alcohol abuse?

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  • 7 years ago
    Favorite Answer

    It depends on many different things. Even though they set up charts to state a

    lethal dose, it does not apply to everyone. It depends on the cause, also.

    Alcohol can cause liver cell damage in different ways:

    1) overconsuming alcohol in a short time period doesn't give the liver cells time

    to make the necessary enzymes to convert the alcohol to acetaldehyde, then

    to acetate, and then to carbon dioxide and water. If the alcohol is not

    converted complete to the last step, it can become toxic to the liver cells.

    2)consuming alcohol over long time periods can lead to fat build up inside

    the liver (fatty liver disease). The fat causes additional pressure inside

    the liver that can also damage the liver cells.

    3)some people are more sensitive to alcohol and some are even allergic

    to it. Alcohol is a drug and just like any other drug...some people may

    have little problem consuming it, while others...it doesn't take much to

    cause one.

    4)mixing alcohol with medication can lead to an interaction occuring

    between them...or the liver is overwhelmed in handling both at the

    same time. That can cause instant cell damage.

    So, it really depends on a lot of different things whether a dosage would

    be lethal or not.

    When the liver cells become damaged, the immune system of the body

    responds to this and cause inflammation inside the liver. This causes

    the liver to enlarge in size and takes on a spongy texture.

    This inflammation, if it continues in the liver and the cause is not

    stopped can lead to where the liver cells die off and form scar tissue

    inside the liver...this is then Fibrosis. When the scar tissue keeps

    forming in the liver, it start to connect together...this is

    bridging Fibrosis. The scar tissue starts to reconstruct the

    liver on the inside and the blood doesn't flow through properly.

    The scar tissue prevents healthy cells and regenerated cells

    from receiving blood and therefore they continue to die off.

    All blood from the entire abdominal area flows through the

    portal vein of the liver, to the hepatic vein and then through

    the vena cava vessel to the heart. The scar tissue also

    prevents this from taking place...the blood backs up

    in the portal vein and other smaller WEAK vessels

    under the liver (varies) that can break open and

    cause internal bleeding. This is now Cirrhosis of

    the liver.

    Being detoxed from alcohol is the very first step in preventing

    further damage to the liver cells. As long as there is just

    inflammation with enlargement...there is a good chance that

    it could be reversed. Once it reaches the stage of scar tissue

    forming, it will become Cirrhosis...a progressive irreversible

    disease. There is a fine line between the two.

    A liver biopsy is the best test known to determine this.

    Most all Transplant centers require a patient who is

    addicted to alcohol, to be detoxed (under doctors care)

    for a period of six month prior to being referred to them

    for the evaluation process of being placed on the liver

    transplant list. It is sad that many don't live to make

    it during that period of time.

    Here is a chart you might want to look at:

    0.02 Slight mood changes.

    0.05-0.06 Impairment starts at .05

    Lowered inhibition, impaired judgement, decreased rational decision making ability.

    0.08 Legally drunk, deterioration of reaction time & control. Driving is dangerous.

    0.10-0.12 Memory and judgment, and motor skills are markedly impaired;

    lack of coordination and balance; recognition of impairment is lost;

    impotence can occur; increasing belligerence in some. Driving is extremely dangerous.

    .15 Impaired balance, movement and coordination. Difficulty standing, walking or talking.

    Blackouts (periods of amnesia for all or part of drinking episode) occur for some at

    the .14-.17 level.

    .20 Decreased pain & sensation. Erratic emotions. Nausea/vomiting may occur.

    Risk for asphyxiation on own vomit.

    .30 Semi-conscious. Diminished reflexes.

    .40 Loss of consciousness (comatose). Very limited reflexes. Anesthetic effects.

    Death possible due to respiratory arrest.

    .50 Deep coma and death due to anesthesia of nerve centers controlling respiration

    and heartbeat.

    >.50 Half the people who reach this level die.

    I hope this information has been of some help to you.

    Best wishes

  • 7 years ago

    Not all that much. Toxicity is a fairly straightfrward thing. .64 is considered lethal whether a first time drinker succumbs at .61 ot as heavy drinker succumbs at .67 isn't critical. The critivcal information is that .6 is a wake up call ...

    Long term ingestion of poisons like ethyl alcohol eventually destroy the liver.The term is cirrhosis. Symptoms of liver failure include yellowing of the eyes and skin.

  • mcgrew
    Lv 4
    3 years ago

    All the place dope suppose that however man i am gonna ought to go along with #5!! "Ya Yaaaaaaaaaaaaa" ***EDIT*** Sorry you said to opt for two so I will even go along with eleven. However #5 is the quality!

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