KALEX
Lv 6
KALEX asked in 社會與文化語言 · 1 decade ago

不太會翻專業英文

Yet, the mechanisms underlying the hyperglycemia-

induced endothelial cell apoptosis involving the

activation of NF-nB is still not well studied. Furthermore,

whether NF-nB has a crosstalk with JNK cascade and

thereby regulates endothelial cell apoptosis remains unclear.

Through activation of the downstream serine/threonine

kinase Akt, phosphatidylinositol 3V-kinase (PI3K) plays an

important role in averting cell death produced by many

death stimuli [17–21].

1 Answer

Rating
  • 婷婷
    Lv 6
    1 decade ago
    Favorite Answer

    Yet, the mechanisms underlying the hyperglycemia-

    但機制高血糖-

    induced endothelial cell apoptosis involving the

    誘導內皮細胞凋亡涉及在

    activation of NF-nB is still not well studied. Furthermore,

    NF 的啟動n還不好研究了 乙。 此外,

    whether NF-nB has a crosstalk with JNK cascade and

    是否 NF-n B 有一個串擾 JNK 級聯與及

    thereby regulates endothelial cell apoptosis remains unclear.

    從而規管內皮細胞凋亡仍然不明確。

    Through activation of the downstream serine/threonine

    通過啟動的下游絲氨酸/蘇氨酸

    kinase Akt, phosphatidylinositol 3V-kinase (PI3K) plays an

    激酶 Akt,磷脂醯肌醇 3V -激酶 (PI3K) 播放一

    important role in averting cell death produced by many

    在防止細胞死亡產生的許多重要的角色

    death stimuli [17–21].

    [17–21] 的死亡刺激。

    • Login to reply the answers
Still have questions? Get your answers by asking now.