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1α,25-Dihydroxyvitamin D3 regulates inducible nitric oxide synthase messenger RNA expression and nitric oxide release in macrophage-like RAW 264.7 cells

1α,25-二羥化維生素D3調節誘導型一氧化氮合成酶之傳訊RNA的表達和一氧化氮的釋放，在巨噬細胞－RAW 264.7細胞

The expression of inducible nitric oxide synthase (iNOS) expression and release of nitric oxide (NO) from macrophages are markedly increased in granulomatous infections. Activation of macrophages 1α-hydroxylase results in an increase of 1α,25-dihydroxyvitamin D3 [1,25(OH)2D3].

肉芽腫性感染的巨噬細胞明顯增加了一氧化氮合成酶（iNOS）的表現及一氧化氮（NO）的釋放，巨噬細胞1α-羥化酶的活化作用導致造成 1α,25二羥化維生素D3 （1,25-雙氫氧基維他素D3）的增加。

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1α,25-Dihydroxyvitamin D3 regulates inducible nitric oxide synthase messenger RNA expression and nitric oxide release in macrophage-like RAW 264.7 cells



1α,25-二羥化維生素D3調節似巨噬細胞－RAW 264.7細胞內誘導型一氧化氮合成酶之傳訊RNA的表達和一氧化氮的釋放


The expression of inducible nitric oxide synthase (iNOS) expression and release of nitric oxide (NO) from macrophages are markedly increased in granulomatous infections. Activation of macrophages 1α-hydroxylase results in an increase of 1α,25-dihydroxyvitamin D3 [1,25(OH)2D3].



肉芽腫性感染明顯增加了巨噬細胞表現一氧化氮合成酶（iNOS）及釋放一氧化氮（NO）. 活化巨噬細胞1α-羥化酶導致1α,25二羥化維生素D3 （1,25-雙氫氧基維他素D3）的增加。

Source(s): 自己，居美多年，四十年生醫研究