就是李 asked in 社會與文化語言 · 1 decade ago

請幫我重整這兩家翻譯軟體所翻譯出來的中文內容

求大家來幫我重整這兩家翻譯軟體所翻譯出來的中文內容,因為我真的無法翻譯出一段像樣的中文出來。

譯言堂的內容:除減少 CVD 的風險之外,一些研究顯示大豆或異黃酮消費之間的倒轉的關係而且乳癌冒著〔3,9] 的危險. 大部份的重要的報告的結果在亞洲人口之中被引導〔9] 和澳洲人〔3]. 雌激素參與原因論藉著促有絲分裂刺激〔10] 主要地乳癌.

那雌激素的致癌效果也被調停過它的可能提供的各種不同的氧化新陳代謝產物持續的發情的活動或板機 DNA 藉由形成 DNA 損害使內轉〔11] 。 在雌激素新陳代謝產物之中, 4- 而且 16 α-hydroxyestrone(OHE1) 被認為是致癌,然而, 2 OHE1 的不是〔12].16 α-OHE1 and 2-OHE1 是二在人類〔12] 中的主要部份新陳代謝產物. 因此,比尿的 2 OHE1 to 16 的α-OHE1(2/16 α比)已經被假定當做乳癌風險和一些證據的 biomarker支持了這一假設〔13] 。 異黃酮已經被顯示增加這 2/16 α比〔14] 。 然而,他們不已經為在外生的雌激素上的他們的影響力被評估那停經後的女人接受。

奇摩的內容:除減少CVD 風險以外, 一些研究顯示了相反關係在大豆或isoflavones 消耗量和乳腺癌風險[ 3,9 之間] 。大多重大結果被報告被舉辦了在亞裔人口[ 9 ] 並且澳大利亞人[ 3 之中] 。女性荷爾蒙被介入在乳腺癌裡原因論主要由有絲分裂的刺激[ 10 ] 。女性荷爾蒙的致癌物質的作用並且被斡旋通過也許提供堅持estrogenic 活動或觸發器DNA 損傷由形成DNA adducts 的它各種各樣的氧化代謝產物[ 11 ] 。在女性荷爾蒙代謝產物, 4 和16 之中? - hydroxyestrone (OHE1) 認為是致癌物質的, 但是, 2-OHE1 不是[ 12 ] 。16?-OHE1 和2-OHE1 是二種主要代謝產物在人[ 12 ] 。所以, 泌尿2-OHE1 比與16?-OHE1 (2/16? 比率) 被假設了作為乳腺癌風險biomarker 並且一些證據支持了這個假說[ 13] 。Isoflavones 被顯示增加2/16? 比率[ 14 ] 。但是, 他們未被評估為他們的對postmenopausal 婦女接受的外生女性荷爾蒙的影響。

Update:

原文的內容:

Update 2:

Besides reducing the risk of CVD, some studies showed

inverse relationships between soy or isoflavones consumption

and breast cancer risk [3,9]. Most of the significant

results reported were conducted among Asian populations

[9] and Australians .

Update 3:

Estrogens are involved in the etiology

of breast cancer primarily by mitogenic stimulation [10]. The

carcinogenic effect of estrogens is also mediated through its

various oxidative metabolites which may provide persistent

estrogenic activity or trigger DNA damage by forming DNA

adducts .

Update 4:

Among the estrogen metabolites, 4- and 16α-

hydroxyestrone (OHE1) are thought to be carcinogenic,

whereas, 2-OHE1 is not [12]. 16α-OHE1 and 2-OHE1 are two

main metabolites in humans .

Update 5:

Therefore, the ratio of

urinary 2-OHE1 to 16α-OHE1 (2/16α ratio) has been hypothesized

as a biomarker of breast cancer risk and some evidence

supported this hypothesis .

Update 6:

Isoflavones have been shown

to increase the 2/16α ratio . However, they have not

been evaluated for their influence on exogenous estrogens

that postmenopausal women receive.

2 Answers

Rating
  • 1 decade ago
    Favorite Answer

    你的原文在哪阿........沒原文我也沒辦法幫你

    2007-11-14 21:44:08 補充:

    Besides reducing the risk of CVD, some studies showed

    inverse relationships between soy or isoflavones consumption

    and breast cancer risk [3,9].

    除了減少CVD 風險以外, 一些研究顯示了大豆或大豆異黃酮的消耗量與乳腺癌風險相對關係[ 3,9] 。

    Most of the significant results reported were conducted among Asian

    populations [9] and Australians .

    絕大部分的研究結果是對亞裔人口[ 9 ] 還有澳大利亞人[ 3 ]測試 。

    Estrogens are involved in the etiology of breast cancer primarily by mitogenic stimulation [10].

    理論上女性荷爾蒙被認為是乳腺癌導因裡主要是因為有絲分裂[ 10 ] 。

    The carcinogenic effect of estrogens is also mediated through its various oxidative metabolites which may provide persistent estrogenic activity or trigger DNA damage by forming DNA adducts .

    女性荷爾蒙的致癌物質以冬眠的方式躲過氧化代謝然後可能幫助 雌激素活化並以粘合DNA的方式照成DNA 損傷由[ 11 ] 。

    Among the estrogen metabolites, 4- and 16α-hydroxyestrone (OHE1)

    are thought to be carcinogenic,whereas, 2-OHE1 is not [12].

    16α-OHE1 and 2-OHE1 are two main metabolites in humans .

    在女性荷爾蒙代謝產物, 4- 和 16 α- hydroxyestrone (OHE1) 被認為是致癌物質, 但是, 2-OHE1 不是[ 12 ] 。16α-OHE1 與 2-OHE1 是二種人體的主要代謝產物[ 12 ] 。

    Therefore, the ratio of urinary 2-OHE1 to 16α-OHE1 (2/16α ratio) has been hypothesized as a biomarker of breast cancer risk and some evidence supported this hypothesis .

    所以, 尿中的比例 2-OHE1 與16 α-OHE1 (2/16α 比例) 被假設為乳腺癌風險的生化黑手並且有一些證據支持了這個假說[ 13] 。

    Isoflavones have been shown to increase the 2/16α ratio . However,

    they have not been evaluated for their influence on exogenous estrogens

    that postmenopausal women receive.

    Isoflavones 可以有效增加2/16α 比率[ 14 ] 。但是, 他們尚未評估

    更年期婦女接受的外來女性荷爾蒙的影響。

    2007-11-14 21:45:24 補充:

    isoflavone

    異黃酮

    2007-11-14 21:46:59 補充:

    你怎嚜贊助自己的提問阿...ㄏㄏ

    Source(s): 真難搞.....不過稿了
  • 小蔡
    Lv 6
    1 decade ago

    Besides reducing the risk of CVD, some studies showed

    inverse relationships between soy or isoflavones consumption

    and breast cancer risk [3,9]. Most of the significant

    results reported were conducted among Asian populations

    [9] and Australians .

    除了減少CVD的危險,一些研究顯示相對關係在大豆或大豆異黃酮消耗量和乳癌危險之間,大多數徵狀結果報告是表現亞洲人口和澳大利人之間。

    Estrogens are involved in the etiology

    of breast cancer primarily by mitogenic stimulation [10]. The

    carcinogenic effect of estrogens is also mediated through its

    various oxidative metabolites which may provide persistent

    estrogenic activity or trigger DNA damage by forming DNA

    adducts .

    女性荷爾蒙是被引起在乳癌初期的原因透過絲分裂。女性荷爾蒙的致癌物也間接透過它的過氧的代謝物可能提供持續生存的雌激素活化或觸發DNA毀壞透過形式的DNA收斂。

    Among the estrogen metabolites, 4- and 16α-

    hydroxyestrone (OHE1) are thought to be carcinogenic,

    whereas, 2-OHE1 is not [12]. 16α-OHE1 and 2-OHE1 are two

    main metabolites in humans .

    在雌激素代謝物─4-和16α烃雌素酮(OHE1)是被想做致癌物的,反之,2-OHE1卻不是。16α-OHE1和2-OHE1是兩個人類主要的代謝物。

    Therefore, the ratio of

    urinary 2-OHE1 to 16α-OHE1 (2/16α ratio) has been hypothesized

    as a biomarker of breast cancer risk and some evidence

    supported this hypothesis .

    因此,尿的比例2-OHE1對16α-OHE1已經是假設化的像乳癌危險和一些跡象的生物指標支持這個假說。

    Isoflavones have been shown

    to increase the 2/16α ratio . However, they have not

    been evaluated for their influence on exogenous estrogens

    that postmenopausal women receive.

    大豆異黃酮已經顯示對增加這2/16α的比例。不管如何,他們已經沒有評價給他們的影響力在更年期過後的女性所吸收外生的(外因的)女性荷爾蒙上。

    Biomarker是作為一個生物學的國家的指標使用的一種物質。

    Source(s): TCC
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